The research team of the German Institute for Nutrition Research (DIfE) has a new relationship between the Dit method of interval fattening and Diabetes Type 2 Diabetes discovered.
Scientists have found that overweight mice that are prone to diabetes have a high accumulation of pancreatic fat cells. In contrast, excessive rodents, which were protected against diabetes due to their genetic makeup, had almost no fat in the pancreatic region.
Interval fast against diabetes – these are the findings of the studies
The researchers decided to divide the diabetic muse into two groups:
- Animals from the first group were allowed to eat as much as they want each day.
- The animals of the other group received interval nutrition: one day the husband got as much food as she wanted, and the next day she had nothing.
After only five weeks, the first differences in the pancreatic area were observed. In the second group, rodents hardly had fatty deposits.
Fat cells in the pancreas as the cause of diabetes
In order to find out what effect the formed fat cells have on the function of the pancreas, scientists have isolated individual fat precursors. After the maturation of the precursor in the fatty cell, increased insulin release could be seen. Insulin is a hormone that regulates blood sugar levels.
Professor Annette Schrmann, study leader, interprets these observations in the following way: "We suspect that increased insulin release will expel Langerhans islands more quickly from diabetic-related animals and ultimately interrupt their function, leading to fat in the pancreas. Contributing to the onset of type 2 diabetes."
Interval post as a future therapeutic approach to diabetes
The study establishes a new relationship between fatty deposits in pancreatic and type 2 diabetes: "Fat accumulation outside of fatty tissue, for example in the liver, muscles or even in bones, has a negative effect on these organs and the whole body. The effect of fat cells inside the pancreas has not been clear until now, "explains Schrmann. Professor Tim J. Schulz, who also contributed to the study and leader of the development and nutrition of fat cells at DifE, suggests: "Accumulation of pancreatic fat tissue can be a key contributor to the development of type 2 diabetes under certain genetic conditions."
Therefore, fasting at intervals can be a promising blood sugar therapy in the near future.
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